By Ace Landers
When Anakin Skywalker and Obi-Wan Kenobi are assigned to guard Senator Padmé Amidala, they determine it will be a simple task. that's, until eventually Obi-Wan falls for a cookie catch and lands them in count number Dooku's enviornment of monster aliens!
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Extra resources for Anakin to the Rescue
Wt1 is a transcriptional activator of the erythropoietin gene. Loss of WT1 expression results in diminished erythropoietin receptor (EpoR) expression in hematopoietic progenitors, suggesting that activation of the EpoR gene by Wt1 is an important mechanism in normal hematopoiesis (Dame et al. 2006). WT1 mutations cause translation of an aberrant protein with loss of normal function and might therefore result in stem cell proliferation and blocking of differentiation, thereby contributing to leukemogenesis (Hollink et al.
Intrathecal chemotherapy for CNS therapy is given during induction and must be continued throughout maintenance therapy. Effective CNS prophylactic regimens have reduced the incidence of isolated CNS relapse to less than 5 %. Hematopoietic stem cell transplant (HSCT) Owing to the associated morbidity and mortality and long-term effects of this procedure, it is reserved for selected children with very high-risk ALL and for some patients who relapse after standard treatment. For induction failure, several leukemia groups recommend allogeneic HSCT; however, it has recently been suggested that some of these children may do well if they receive additional chemotherapy rather than a stem cell transplant.
This translocation leads to the fusion of the retinoic acid receptor α (RARA) to various partner genes. The genes involved are PML, located on chromosome 15, and the RARA gene on chromosome 17, which form the fusion PML-RARA gene expressed exclusively in the APL subtype. The normal RARA is a transcription factor involved in the differentiation of myeloid cells; it needs all-trans retinoic acid (ATRA) (its ligand) to transactivate genes. The resultant RARA chimeric oncoproteins are involved in the pathogenesis of the APL and contribute to leukemic transformation by dominant inhibition of the expression 1 Introduction: Childhood Leukemia 27 of target genes that are important for cellular differentiation.